[{"name": "non-essential genes", "count": 3811, "desc": "Georgi B., et al. From mouse to human: evolutionary genomics analysis of human orthologs of essential genes. PLos Genetics (2013)"}, {"name": "essential genes", "count": 2472, "desc": "Georgi B., et al. From mouse to human: evolutionary genomics analysis of human orthologs of essential genes. PLos Genetics (2013)"}, {"name": "CHD8 target genes", "count": 2158, "desc": "Cotney J, et al. The autism-associated chromatin modifier CHD8 regulates other autism risk genes during human neurodevelopment. Nat Commun. (2015)"}, {"name": "brain embryonically expressed", "count": 1912, "desc": "Iossifov I., et al. The contribution of de novo coding mutations to autism spectrum disorder. Nature (2014)"}, {"name": "brain critical genes", "count": 1744, "desc": "Uddin M, et al. Brain-expressed exons under purifying selection are enriched for de novo mutations in autism spectrum disorder. Nat Genetics (2014)"}, {"name": "PSD", "count": 1445, "desc": "Bayes A., et al. Characterization of the proteome, diseases and evolution of the human postsynaptic density. Nature Neuroscience (2011)"}, {"name": "FMRP Tuschl", "count": 939, "desc": "Ascano M., et al. FMRP targets distinct mRNA sequence elements to regulate protein expression. Nature (2012)"}, {"name": "FMRP Darnell", "count": 842, "desc": "Darnell JC., et al. FMRP stalls ribosomal translocation on mRNAs linked to synaptic function and autism. Cell (2011)"}, {"name": "chromatin modifiers", "count": 428, "desc": "Iossifov I., et al. The contribution of de novo coding mutations to autism spectrum disorder. Nature (2014)"}, {"name": "synaptic clefts excitatory", "count": 194, "desc": "Ken H. Loh, et al. Proteomic Analysis of Unbounded Cellular Compartments: Synaptic Clefts. Cell (2016)"}, {"name": "postsynaptic inhibition", "count": 181, "desc": "Akiyoshi Uezu, et al. Identification of an elaborate complex mediating postsynaptic inhibition. Science (2016)"}, {"name": "topotecan downreg genes", "count": 154, "desc": "King IF, et al. Topoisomerases facilitate transcription of long genes linked to autism. Nature (2013)"}, {"name": "synaptic clefts inhibitory", "count": 41, "desc": "Ken H. Loh, et al. Proteomic Analysis of Unbounded Cellular Compartments: Synaptic Clefts. Cell (2016)"}]